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Mannitol

Osmotic diuretic

CCP: Reduction of intracranial pressure and cerebral edema

  • Patients with well-established anuria as a result of severe renal disease and who do not respond to 2 test doses
  • Severe pulmonary congestion or frank pulmonary edema
  • Severe congestive heart failure
  • Dehydration states
  • Metabolic edema associated with capillary fragility or membrane permeability
  • Progressive renal disease

CCP: Reduction of intracranial pressure and cerebral edema

  • 1.5-2 g/kg IV infused as a 15%, 20%, or 25% solution 
  • 0.25 g/kg IV not more frequently than every 6-8 hours

CCP: Reduction of intracranial pressure and cerebral edema

  • 2 g/kg IV infused as a 15% or 20% solution

Intravenous solution: 200 mg/mL in 500 mL bag of 20% solution

Mannitol increases extracellular fluid volume and dilutes extracellular stores of sodium, drawing water out of the cells into the plasma.  Fluid shifts result in the reduction of cerebral edema and lowering of cerebrospinal fluid pressure. 

Intravenous:

  • CSF pressure reduced within 15 minutes
  • Diuresis after 1-3 hours
  • Intraocular pressure reduced within 30-60 minutes

Mannitol use may disturb other fluid and electrolyte balances

Accumulation of mannitol caused by inadequate urinary output, or rapid adminsitration of large volumes, may result in the overexpansion of extracellular fluid and circulatory overload causing signs and symptoms of water intoxication.  Overhydration may be corrected by hemodialysis or administration of a diuretic. 

There is a risk of serious electrolyte disturbances, which may be severe enough to alter the acid-base balance, or to depress respirations.  Thiazides may be used if hypernatremia or hyperosmolality occurs.

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